AKR1C4 Antibody (N-term) Blocking Peptide
€363.00
In stock
SKU
AC-BP16366a
Background:
AKR1C4 is a member of the aldo/keto reductasesuperfamily, which consists of more than 40 known enzymes andproteins. These enzymes catalyze the conversion of aldehydes andketones to their corresponding alcohols by utilizing NADH and/orNADPH as cofactors. The enzymes display overlapping but distinctsubstrate specificity. This enzyme catalyzes the bioreduction ofchlordecone, a toxic organochlorine pesticide, to chlordeconealcohol in liver. This gene shares high sequence identity withthree other gene members and is clustered with those three genes atchromosome 10p15-p14.
Other Names:
Aldo-keto reductase family 1 member C4, 111-, 3-alpha-HSD1, 3-alpha-hydroxysteroid dehydrogenase type I, Chlordecone reductase, CDR, Dihydrodiol dehydrogenase 4, DD-4, DD4, HAKRA, AKR1C4, CHDR
Gene Name: AKR1C4
Gene ID: 1109
Primary Accession: P17516
Format: Peptides are lyophilized in a solid powder format. Peptides can be reconstituted in solution using the appropriate buffer as needed.
AKR1C4 is a member of the aldo/keto reductasesuperfamily, which consists of more than 40 known enzymes andproteins. These enzymes catalyze the conversion of aldehydes andketones to their corresponding alcohols by utilizing NADH and/orNADPH as cofactors. The enzymes display overlapping but distinctsubstrate specificity. This enzyme catalyzes the bioreduction ofchlordecone, a toxic organochlorine pesticide, to chlordeconealcohol in liver. This gene shares high sequence identity withthree other gene members and is clustered with those three genes atchromosome 10p15-p14.
Other Names:
Aldo-keto reductase family 1 member C4, 111-, 3-alpha-HSD1, 3-alpha-hydroxysteroid dehydrogenase type I, Chlordecone reductase, CDR, Dihydrodiol dehydrogenase 4, DD-4, DD4, HAKRA, AKR1C4, CHDR
Gene Name: AKR1C4
Gene ID: 1109
Primary Accession: P17516
Format: Peptides are lyophilized in a solid powder format. Peptides can be reconstituted in solution using the appropriate buffer as needed.
| Is Featured? | No |
|---|
Write Your Own Review